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    <loc>https://www.thereversalproject.com/blog/blog-post-title-one-37ek8</loc>
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      <image:title>Blog - Insulin&lt;/span&gt; - Diabetes Mellitus</image:title>
      <image:caption>The term “diabetes” predates the currently known pathophysiology. In Greek, “diabetes” literally means “to go through,” which refers to the excessive urination characteristic of all forms of diabetes. Diabetes mellitus is what is typically referred to as “diabetes”. “Mellitus” is Greek for “sweet”. When your blood sugar gets too high, your body excretes excess sugar that it is unable to absorb through the urine. Both type I and type II diabetes mellitus were characterized as the same or similar diseases because they both involved excessive glucose in the urine. However, the pathophysiology of these diseases is markedly different.</image:caption>
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      <image:title>Blog - Insulin&lt;/span&gt; - Type I Diabetes</image:title>
      <image:caption>Type 1 diabetes is often referred to as “juvenile diabetes” by non-medical professionals. Medicine no longer uses this terminology, and for good reason. Type 1 diabetes can develop at any time in the lifespan. And today, type II diabetes is more common in children than Type I diabetes (though in generations past, type II diabetes in children was exceedingly rare). Type I is usually caused by an autoimmune response or viral infection that attacks the pancreas, the organ that secretes insulin (in addition to digestive enzymes and other hormones).</image:caption>
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      <image:title>Blog - Insulin&lt;/span&gt; - Type II Diabetes</image:title>
      <image:caption>Unlike type I diabetes, there is no shortage of insulin in type II diabetes. Rather than low insulin levels, type II diabetes is characterized by insulin resistance, a complex condition in which cells in the body do not respond to insulin as they should. Cells in your muscles, liver, and fat uptake less insulin and blood sugar (they ‘resist’ letting insulin inside), which leads to a buildup of glucose in the blood. In response to elevated blood glucose, the pancreas secretes more insulin, which leads to greater insulin resistance and higher blood glucose levels.</image:caption>
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      <image:title>Blog - Insulin&lt;/span&gt; - Make it stand out</image:title>
      <image:caption>Whatever it is, the way you tell your story online can make all the difference.</image:caption>
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      <image:title>Blog - Insulin&lt;/span&gt; - Protein</image:title>
      <image:caption>Protein causes a very small increase in insulin secretion. However, it will not be the rollercoaster ride of blood glucose and insulin that you get from carbohydrates. Proteins take more energy to digest than any other macronutrient, and they are also more satiating than any other macronutrient. When you plan your meals, protein should be the star of the show.</image:caption>
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      <image:title>Blog - Insulin&lt;/span&gt; - Fat</image:title>
      <image:caption>Fat causes no increase in insulin or blood sugar. Fat is also incredibly satiating, and a small amount of fat is required to absorb many micronutrients. Note that fat is the most calorie-dense macronutrient. Though fat is necessary to sustain life, it can be consumed in excess, leading to extra calories and weight gain. Measure your oils, nuts, and butter.</image:caption>
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      <image:title>Blog - Insulin&lt;/span&gt; - Medications</image:title>
      <image:caption>As you can imagine, injecting yourself with insulin from a vial will not lower your insulin and help you lose weight. It will raise your insulin levels, make your insulin resistance worse, and cause weight gain. There are other medications that do help combat insulin resistance. Keep in mind, all of these medications are prescription only. If you want medication therapy to help manage your insulin resistance, you will need to talk to your doctor.</image:caption>
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      <image:title>Blog - Insulin&lt;/span&gt; - Exercise</image:title>
      <image:caption>Muscles need energy to function. The more muscle tissue you have, the more energy your muscles will need. When you have excess blood sugar after a meal, one of the first places your body sends the blood glucose is the muscle tissue. The more you work your muscles, the more sugar from the blood will have to replace the sugar your muscles use.  Consistency is key. Just 20 minutes of activity a day will yield more results than being a gym rat for a few weeks before you hurt yourself and give up. Resistance training to prevent muscle loss as you age will help prevent age-related metabolic slowdown.</image:caption>
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      <image:title>Blog - Insulin&lt;/span&gt; - Weight Loss Surgery</image:title>
      <image:caption>Weight-loss surgeries are among the most drastic weight-loss options and involve weeks to months of recovery. Typically, weight-loss surgery involves reducing stomach capacity to increase satiety, and more extreme procedures also bypass the upper small intestine, which is responsible for nutrient absorption. Often, the extreme reduction in calories results in type II diabetes reversal within a week of surgery. Just like with medications, you can “out eat” your weight loss surgery.</image:caption>
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      <image:title>Blog - Polycystic Ovarian Syndrome&lt;/span&gt; - OK… Time for an A&amp;P Lesson.</image:title>
      <image:caption>Ovaries are organs in the body that produce eggs and the female hormones, progesterone and estrogen. In PCOS, the ovaries produce more androgens than they should. Androgens are male sex hormones, such as testosterone. This can lead women with PCOS to have increased body and facial hair growth, acne and oily skin, a deeper voice, male pattern baldness, and other masculine characteristics. This often causes psychological distress and can lead to anxiety, depression, and body image issues.</image:caption>
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      <image:title>Blog - Polycystic Ovarian Syndrome&lt;/span&gt; - Keep in mind: this was all simplified for brevity. If you want to get into the nuances on your own, they are quite fascinating.</image:title>
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    <loc>https://www.thereversalproject.com/blog/blog-post-title-three-xs7pp</loc>
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      <image:title>Blog - Diabetes is Costly&lt;/span&gt; - When The Cost Becomes Astronomical Unmanaged diabetes progresses and worsens over time, and often leads to further complications.</image:title>
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      <image:title>Blog - Diabetes is Costly&lt;/span&gt; - A Better Option: The Reversal Project</image:title>
      <image:caption>Type II diabetes is always preventable and always reversible. No medication is necessary. You can lower your blood sugar and insulin resistance with diet and lifestyle changes. The Reversal Project provides education rooted in current science and best practices. We work with patients, primary care providers, and endocrinologists to implement an evidence-based lifestyle intervention. This includes tailored nutrition and activity plans, coaching and support from experts, and access to a community of health-driven patients. The best part: The Reversal Project does this for just a fraction of the cost of your diabetes care.</image:caption>
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    <loc>https://www.thereversalproject.com/blog/blog-post-title-four-hrssl</loc>
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      <image:title>Blog - Hunger Hormones&lt;/span&gt; - Let's start with the two major hunger hormones: Leptin and Ghrelin</image:title>
      <image:caption>Ghrelin Ghrelin can be thought of as the “hunger hormone”. It is primarily produced in the stomach, but also produced by the small intestines, brain, and pancreas. When your stomach is empty, it sends a powerful signal to your brain that it is time to eat. You feel this as hunger. About 20 minutes after your stomach is full, it stops producing ghrelin.  Pro tip: Use this information to your advantage. It takes 20 minutes to feel full. You could shovel down food as fast as possible until you are stuffed. OR… You could eat slowly and pause throughout your meal. Give ghrelin a chance to start circulating through your body.</image:caption>
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      <image:title>Blog - Hunger Hormones&lt;/span&gt; - Those are the Big Three. There are a Few Others Cholecystokinin</image:title>
      <image:caption>Cholecystokinin is mostly produced in the small intestine in response to eating a meal, and more cholecystokinin is produced when you eat a high-fat meal. Cholecystokinin stimulates the pancreas to produce digestive enzymes, which help you break down food in the small intestine. It also stimulates the gallbladder to release bile, which emulsifies fats in your GI tract so that they can be absorbed. Cholecystokinin also delays gastric emptying, thereby prolonging satiety, and suppresses hunger. Studies indicate that increased amounts of cholecystokinin are associated with decreased appetite</image:caption>
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